Fragment-Based Drug Discovery: Difference between revisions

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The development of <scene name='Sandbox_reserved_394/Abt-737/5'>ABT-737</scene> using SAR by NMR is a classic example of FBDD. (Throughout this discussion ABT-737 will be used to illustrate the FBDD process.) This compound has been shown to effectively inhibit the over-expression of <scene name='Sandbox_reserved_394/Bcl-xl/1'>Bcl-xl</scene> which is a protein that is commonly observed to be over-expressed in many types of cancers.<ref name="Oltersdorf T., Elmore S. W., Shoemaker A. R. An inhibitor of Bcl-2 family proteins induces regression of solid tumours. Vol 435|2 June 2005|doi:10.1038/nature03579">Oltersdorf T., Elmore S. W., Shoemaker A. R. An inhibitor of Bcl-2 family proteins induces regression of solid tumours. Vol 435|2 June 2005|doi:10.1038/nature03579</ref> It acts an inhibitor of apoptosis and may also contribute to chemotherapy resistance. Bcl-xl inhibition by ABT-737 therefore, allows apoptosis to occur and helps to prevent chemo-resistance.
The development of <scene name='Sandbox_reserved_394/Abt-737/6'>ABT-737</scene> using SAR by NMR is a classic example of FBDD. (Throughout this discussion ABT-737 will be used to illustrate the FBDD process.) This compound has been shown to effectively inhibit the over-expression of <scene name='Sandbox_reserved_394/Bcl-xl/1'>Bcl-xl</scene> which is a protein that is commonly observed to be over-expressed in many types of cancers.<ref name="Oltersdorf T., Elmore S. W., Shoemaker A. R. An inhibitor of Bcl-2 family proteins induces regression of solid tumours. Vol 435|2 June 2005|doi:10.1038/nature03579">Oltersdorf T., Elmore S. W., Shoemaker A. R. An inhibitor of Bcl-2 family proteins induces regression of solid tumours. Vol 435|2 June 2005|doi:10.1038/nature03579</ref> It acts an inhibitor of apoptosis and may also contribute to chemotherapy resistance. Bcl-xl inhibition by ABT-737 therefore, allows apoptosis to occur and helps to prevent chemo-resistance.
{| class="wikitable collapsible"
{| class="wikitable collapsible"
! scope="col" width="5000px" | SAR by NMR
! scope="col" width="5000px" | SAR by NMR
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===== ABT-737: ligand screening =====
===== ABT-737: ligand screening =====


<scene name='Sandbox_reserved_394/Compound_1/12'>Two fragments</scene> were found to have moderate affinity for Bcl-xl. <scene name='Sandbox_reserved_394/Compound_1/9'>Compound 1</scene> is a fluorobiphenylcarboxylic acid. It occupies <scene name='Sandbox_reserved_394/Binding_site_1/2'>binding site 1</scene> of Bcl-xl which consists of Phe 101, Tyr 105, Ala 108, Phe 109, Leu 136, Gly 142, Arg 143, and Ala 146. The fluorobiphenyl system of compound 1 is very hydrophobic and therefore, these residues form a <scene name='Sandbox_reserved_394/Compound_1/4'>"hydrophobic pocket"</scene> around the system. There is also one hydrophilic interaction involved in this complex. The <scene name='Sandbox_reserved_394/Compound_1/5'>carboxylic acid portion of compound 1 binds near Gly 142</scene> of Bcl-xl. This is not a strong interaction but is significant because it can be modified to form a much stronger bond.
<scene name='Sandbox_reserved_394/Compound_1/12'>Two fragments</scene> were found to have moderate affinity for Bcl-xl. <scene name='Sandbox_reserved_394/Compound_1/9'>Compound 1</scene> is a fluorobiphenylcarboxylic acid. It occupies <scene name='Sandbox_reserved_394/Binding_site_1/4'>binding site 1</scene> of Bcl-xl which consists of Phe 101, Tyr 105, Ala 108, Phe 109, Leu 136, Gly 142, Arg 143, and Ala 146. The fluorobiphenyl system of compound 1 is very hydrophobic and therefore, these residues form a <scene name='Sandbox_reserved_394/Compound_1/4'>"hydrophobic pocket"</scene> around the system. There is also one hydrophilic interaction involved in this complex. The <scene name='Sandbox_reserved_394/Compound_1/5'>carboxylic acid portion of compound 1 binds near Gly 142</scene> of Bcl-xl. This is not a strong interaction but is significant because it can be modified to form a much stronger bond.


<scene name='Sandbox_reserved_394/Compound_1/3'>Compound 2</scene> is a napthalene-based alcohol which occupies <scene name='Sandbox_reserved_394/Binding_site_2/4'>binding site 2</scene>. This particular fragment also is involved with hydrophobic interactions with Bcl-xl, although they are not as strong as in the case of compound 1. This binding site includes Ala 97, Glu 100, Phe 101, Val 145, and Tyr 199.
<scene name='Sandbox_reserved_394/Compound_1/3'>Compound 2</scene> is a napthalene-based alcohol which occupies <scene name='Sandbox_reserved_394/Binding_site_2/4'>binding site 2</scene>. This particular fragment also is involved with hydrophobic interactions with Bcl-xl, although they are not as strong as in the case of compound 1. This binding site includes Ala 97, Glu 100, Phe 101, Val 145, and Tyr 199.
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! scope="col" width="5000px" | Modifying compound 3 to reduce HSA affinity
! scope="col" width="5000px" | Modifying compound 3 to reduce HSA affinity
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| scope="col" width="5000px" | Compound 3 has high affinity for Bcl-xl but has an even higher affinity for HSA. For this reason, when HSA is present, compound 3 and similar ligands are more likely to bind to HSA thereby decreasing the amount that can bind with Bcl-xl. In order to decrease the affinity for HSA while maintaining affinity for Bcl-xl, SAR by NMR was used to compare compound 3 with a <scene name='Sandbox_reserved_394/Compound_3/1'>compound 4</scene> (thioethylamino-2,4-dimethylphenyl analogue), which also has high affinity for HSA. It was found that <scene name='Sandbox_reserved_394/Compound_3/2'>two hydrophobic portions</scene> of compound 3 had very strong hydrophobic interactions with HSA. Therefore, these portions were modified with polar substituents to decrease HSA affinity. To decrease hydrophobicity, the fluorobiphenyl system was substituted with a <scene name='Sandbox_reserved_394/Piperazine_ring/2'>piperazine ring</scene> and a <scene name='Sandbox_reserved_394/Abt-737/4'>2-dimethylaminoethyl group</scene> was added to the thioethylamino linkage group.
| scope="col" width="5000px" | Compound 3 has high affinity for Bcl-xl but has an even higher affinity for HSA. For this reason, when HSA is present, compound 3 and similar ligands are more likely to bind to HSA thereby decreasing the amount that can bind with Bcl-xl. In order to decrease the affinity for HSA while maintaining affinity for Bcl-xl, SAR by NMR was used to compare compound 3 with a <scene name='Sandbox_reserved_394/Compound_3/1'>compound 4</scene> (thioethylamino-2,4-dimethylphenyl analogue), which also has high affinity for HSA. It was found that <scene name='Sandbox_reserved_394/Compound_3/3'>two hydrophobic portions</scene> of compound 4 had very strong hydrophobic interactions with HSA. Therefore, these portions in compound 3 were modified with polar substituents to decrease HSA affinity. To decrease hydrophobicity, the fluorobiphenyl system was substituted with a <scene name='Sandbox_reserved_394/Piperazine_ring/2'>piperazine ring</scene> and a <scene name='Sandbox_reserved_394/Abt-737/4'>2-dimethylaminoethyl group</scene> was added to the thioethylamino linkage group.
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Latest revision as of 19:46, 25 June 2013

Drug Design: Fragment-Based Drug Discovery

Bcl-xl in complex with ABT-737 (PDB entry 2yxj)

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References

Proteopedia Page Contributors and Editors (what is this?)

Justin Weekley, Arthur Cox, Jaime Prilusky