Sandbox Reserved 479: Difference between revisions
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==Medical Implications and Possible Applications== | ==Medical Implications and Possible Applications== | ||
The discovery of myosin structure served as a crucial component in understanding the causes of many health related problems. Dysfunctional myosin mutations have been linked to hypertrophy in the heart. The heart enlarges to compensate for inadequate cardiac muscle contractions and consequent poor blood circulation. Also, blood platelet contractile protein aggregation and secretion is impeded by faulty myosin in patients suffering from idiopathic scoliosis. A defect in the less-well known myosin VIIA causes Usher 1B | The discovery of myosin structure served as a crucial component in understanding the causes of many health related problems. Dysfunctional myosin mutations have been linked to hypertrophy in the heart. The heart enlarges to compensate for inadequate cardiac muscle contractions and consequent poor blood circulation. Also, blood platelet contractile protein aggregation and secretion is impeded by faulty myosin in patients suffering from idiopathic scoliosis. A defect in the less-well known myosin VIIA causes Usher 1B syndrome; causing a loss of sight and hearing impairments. Further research will hopefully give more insight into the unknown causes of other diseases and disorders linked to myosin protein mutations.<ref name="CHEM43" /> | ||
==References== | ==References== | ||
<references/> | <references/> | ||
