1f1d: Difference between revisions

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[[Image:1f1d.png|left|200px]]
{{STRUCTURE_1f1d|  PDB=1f1d  |  SCENE=  }}  
{{STRUCTURE_1f1d|  PDB=1f1d  |  SCENE=  }}  
===Crystal structure of yeast H46C cuznsod mutant===
===Crystal structure of yeast H46C cuznsod mutant===
{{ABSTRACT_PUBMED_10026301}}


{{ABSTRACT_PUBMED_10873471}}
==Function==
[[http://www.uniprot.org/uniprot/SODC_YEAST SODC_YEAST]] Destroys radicals which are normally produced within the cells and which are toxic to biological systems.


==About this Structure==
==About this Structure==
[[1f1d]] is a 1 chain structure with sequence from [http://en.wikipedia.org/wiki/Saccharomyces_cerevisiae Saccharomyces cerevisiae]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1F1D OCA].  
[[1f1d]] is a 1 chain structure with sequence from [http://en.wikipedia.org/wiki/Atcc_18824 Atcc 18824]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1F1D OCA].  


==See Also==
==See Also==
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==Reference==
==Reference==
<ref group="xtra">PMID:010873471</ref><references group="xtra"/>
<ref group="xtra">PMID:010026301</ref><references group="xtra"/><references/>
[[Category: Saccharomyces cerevisiae]]
[[Category: Atcc 18824]]
[[Category: Superoxide dismutase]]
[[Category: Superoxide dismutase]]
[[Category: Eisenberg, D.]]
[[Category: Eisenberg, D.]]

Revision as of 07:41, 5 February 2014

Template:STRUCTURE 1f1d

Crystal structure of yeast H46C cuznsod mutant

Template:ABSTRACT PUBMED 10026301

Function

[SODC_YEAST] Destroys radicals which are normally produced within the cells and which are toxic to biological systems.

About this Structure

1f1d is a 1 chain structure with sequence from Atcc 18824. Full crystallographic information is available from OCA.

See Also

Reference

  1. Hart PJ, Balbirnie MM, Ogihara NL, Nersissian AM, Weiss MS, Valentine JS, Eisenberg D. A structure-based mechanism for copper-zinc superoxide dismutase. Biochemistry. 1999 Feb 16;38(7):2167-78. PMID:10026301 doi:https://dx.doi.org/10.1021/bi982284u

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