Sandbox Reserved 1176: Difference between revisions
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===Leptin Research=== | ===Leptin Research=== | ||
NTSR1 deficient mice were not able to receive a '''[https://en.wikipedia.org/wiki/Hunger_(motivational_state) satiety]''' signal.<ref name="Mice"/>The mice continued to eat when food was present, leading to significant weight gain. | NTSR1 deficient mice were not able to receive a '''[https://en.wikipedia.org/wiki/Hunger_(motivational_state) satiety]''' signal from '''[https://en.wikipedia.org/wiki/Leptin Leptin]'''<ref name="Mice"/>. The mice continued to eat when food was present, leading to significant weight gain. The relationship between satiety and NTS is due to the role of NTSR1 in the leptin signaling pathway, and without sufficient NTSR1 this pathway is interrupted. <ref name="Mice"/> | ||
===Cancer Studies=== | ===Cancer Studies=== | ||
