6f2r: Difference between revisions
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The entry | ==A hetrotetramer of human HspB2 and HspB3== | ||
<StructureSection load='6f2r' size='340' side='right' caption='[[6f2r]], [[Resolution|resolution]] 3.90Å' scene=''> | |||
== Structural highlights == | |||
<table><tr><td colspan='2'>[[6f2r]] is a 21 chain structure. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6F2R OCA]. For a <b>guided tour on the structure components</b> use [http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6F2R FirstGlance]. <br> | |||
</td></tr><tr id='NonStdRes'><td class="sblockLbl"><b>[[Non-Standard_Residue|NonStd Res:]]</b></td><td class="sblockDat"><scene name='pdbligand=UNK:UNKNOWN'>UNK</scene></td></tr> | |||
[[Category: | <tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6f2r FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6f2r OCA], [http://pdbe.org/6f2r PDBe], [http://www.rcsb.org/pdb/explore.do?structureId=6f2r RCSB], [http://www.ebi.ac.uk/pdbsum/6f2r PDBsum], [http://prosat.h-its.org/prosat/prosatexe?pdbcode=6f2r ProSAT]</span></td></tr> | ||
</table> | |||
== Disease == | |||
[[http://www.uniprot.org/uniprot/HSPB3_HUMAN HSPB3_HUMAN]] Distal hereditary motor neuropathy type 2. The disease is caused by mutations affecting the gene represented in this entry. | |||
== Function == | |||
[[http://www.uniprot.org/uniprot/HSPB2_HUMAN HSPB2_HUMAN]] May regulate the kinase DMPK.<ref>PMID:9490724</ref> [[http://www.uniprot.org/uniprot/HSPB3_HUMAN HSPB3_HUMAN]] Inhibitor of actin polymerization. | |||
== References == | |||
<references/> | |||
__TOC__ | |||
</StructureSection> | |||
[[Category: Boelens, W C]] | |||
[[Category: Clark, A R]] | |||
[[Category: Cole, A R]] | |||
[[Category: Keep, N H]] | |||
[[Category: Slingsby, C]] | |||
[[Category: Chaperone]] | |||
[[Category: Crystallin]] | |||
[[Category: Shsp]] | |||
Revision as of 07:22, 25 July 2018
A hetrotetramer of human HspB2 and HspB3
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