1hph: Difference between revisions

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~~[[Image:1hph.png|left|200px]]~~

~~<!--~~

~~The line below this paragraph, containing "STRUCTURE_1hph", creates the "Structure Box" on the page.~~

~~You may change the PDB parameter (which sets the PDB file loaded into the applet)~~

~~or the SCENE parameter (which sets the initial scene displayed when the page is loaded),~~

~~or leave the SCENE parameter empty for the default display.~~

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===STRUCTURE OF HUMAN PARATHYROID HORMONE 1-37 IN SOLUTION===

==Disease==

[[http://www.uniprot.org/uniprot/PTHY_HUMAN PTHY_HUMAN]] Defects in PTH are a cause of familial isolated hypoparathyroidism (FIH) [MIM:[http://omim.org/entry/146200 146200]]; also called autosomal dominant hypoparathyroidism or autosomal dominant hypocalcemia. FIH is characterized by hypocalcemia and hyperphosphatemia due to inadequate secretion of parathyroid hormone. Symptoms are seizures, tetany and cramps. FIH exist both as autosomal dominant and recessive forms of hypoparathyroidism.<ref>PMID:2212001</ref><ref>PMID:10523031</ref><ref>PMID:18056632</ref>

~~<!--~~

==Function==

~~The line below this paragraph, {{ABSTRACT_PUBMED_7797503}}, adds the Publication Abstract to the page~~

[[http://www.uniprot.org/uniprot/PTHY_HUMAN PTHY_HUMAN]] PTH elevates calcium level by dissolving the salts in bone and preventing their renal excretion. Stimulates [1-14C]-2-deoxy-D-glucose (2DG) transport and glycogen synthesis in osteoblastic cells.<ref>PMID:21076856</ref>

~~(as it appears on PubMed at~~ http://www.~~pubmed~~.~~gov), where 7797503 is~~ the ~~PubMed ID number~~.

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~~{{ABSTRACT_PUBMED_7797503}}~~

==About this Structure==

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==Reference==

[[Category: Homo sapiens]]

[[Category: Marx, U C.]]

[[Category: Roesch, P.]]

[[Category: Hormone]]

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@@ Line 1: / Line 1: @@
-[[Image:1hph.png|left|200px]]
-<!--
-The line below this paragraph, containing "STRUCTURE_1hph", creates the "Structure Box" on the page.
-You may change the PDB parameter (which sets the PDB file loaded into the applet)
-or the SCENE parameter (which sets the initial scene displayed when the page is loaded),
-or leave the SCENE parameter empty for the default display.
--->
 {{STRUCTURE_1hph|  PDB=1hph  |  SCENE=  }}
 ===STRUCTURE OF HUMAN PARATHYROID HORMONE 1-37 IN SOLUTION===
+{{ABSTRACT_PUBMED_7797503}}
+==Disease==
+[[http://www.uniprot.org/uniprot/PTHY_HUMAN PTHY_HUMAN]] Defects in PTH are a cause of familial isolated hypoparathyroidism (FIH) [MIM:[http://omim.org/entry/146200 146200]]; also called autosomal dominant hypoparathyroidism or autosomal dominant hypocalcemia. FIH is characterized by hypocalcemia and hyperphosphatemia due to inadequate secretion of parathyroid hormone. Symptoms are seizures, tetany and cramps. FIH exist both as autosomal dominant and recessive forms of hypoparathyroidism.<ref>PMID:2212001</ref><ref>PMID:10523031</ref><ref>PMID:18056632</ref>
-<!--
+==Function==
-The line below this paragraph, {{ABSTRACT_PUBMED_7797503}}, adds the Publication Abstract to the page
+[[http://www.uniprot.org/uniprot/PTHY_HUMAN PTHY_HUMAN]] PTH elevates calcium level by dissolving the salts in bone and preventing their renal excretion. Stimulates [1-14C]-2-deoxy-D-glucose (2DG) transport and glycogen synthesis in osteoblastic cells.<ref>PMID:21076856</ref>
-(as it appears on PubMed at http://www.pubmed.gov), where 7797503 is the PubMed ID number.
--->
-{{ABSTRACT_PUBMED_7797503}}
 ==About this Structure==
@@ Line 22: / Line 13: @@
 ==Reference==
-<ref group="xtra">PMID:007797503</ref><references group="xtra"/>
+<ref group="xtra">PMID:007797503</ref><references group="xtra"/><references/>
 [[Category: Homo sapiens]]
 [[Category: Marx, U C.]]
 [[Category: Roesch, P.]]
 [[Category: Hormone]]