Sandbox Reserved 596: Difference between revisions

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In 2001 Kolbelt et al. studied ischemic cerebrovascular disease (a thrombotic disorder) and found that chronic stroke victims showed high plasma concentrations of PZ but just a few months later Vasse et al. found that ischemic stroke patients had low levels of PZ. Shortly after that in 2002 Lopaciuk et al. found no correlation between PZ levels and chronic stroke and Heeb et al. found low levels associated with stroke but not in women and diabetics.

In 2002 Gris et al. examined PZ levels in pathologic pregnancies and found that low PZ levels caused extreme insufficiency of the placenta following the mother/fetus circulatory connection. Low PZ levels were also linked to antiphospholipid antibodies syndrome (aPL), suggesting PZ-ZPI complex incompetency by Steffano et al. and confirmed by a later study by McColl et al. Kemkes-Matthes et al. found that patients with thromboembolic episodes and factor V Leiden mutations suffered greatly from early thrombosis due to PZ deficiency. Lower levels of plasma PZ have also been linked to chronic inflammatory diseases like Bençet’s disease and ischemic colitis, and acute coronary syndromes (ACS).

In 2002 Gris et al. examined PZ levels in pathologic pregnancies and found that low PZ levels caused extreme insufficiency of the placenta following the mother/fetus circulatory connection. Low PZ levels were also linked to antiphospholipid antibodies syndrome (aPL), suggesting PZ-ZPI complex incompetency by Steffano et al. and confirmed by a later study by McColl et al. Kemkes-Matthes et al. found that patients with thromboembolic episodes and factor V Leiden mutations suffered greatly from early thrombosis due to PZ deficiency. Lower levels of plasma PZ have also been linked to chronic inflammatory diseases like Bençet’s disease and ischemic colitis, and acute coronary syndromes (ACS). [1,6]

Sandbox Reserved 596: Difference between revisions

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Revision as of 18:56, 22 April 2013 view source Student (talk \| contribs) students 33,923 edits No edit summary ← Older edit		Revision as of 18:57, 22 April 2013 view source Student (talk \| contribs) students 33,923 edits No edit summary Newer edit →
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	In 2001 Kolbelt et al. studied ischemic cerebrovascular disease (a thrombotic disorder) and found that chronic stroke victims showed high plasma concentrations of PZ but just a few months later Vasse et al. found that ischemic stroke patients had low levels of PZ. Shortly after that in 2002 Lopaciuk et al. found no correlation between PZ levels and chronic stroke and Heeb et al. found low levels associated with stroke but not in women and diabetics.		In 2001 Kolbelt et al. studied ischemic cerebrovascular disease (a thrombotic disorder) and found that chronic stroke victims showed high plasma concentrations of PZ but just a few months later Vasse et al. found that ischemic stroke patients had low levels of PZ. Shortly after that in 2002 Lopaciuk et al. found no correlation between PZ levels and chronic stroke and Heeb et al. found low levels associated with stroke but not in women and diabetics.

	In 2002 Gris et al. examined PZ levels in pathologic pregnancies and found that low PZ levels caused extreme insufficiency of the placenta following the mother/fetus circulatory connection. Low PZ levels were also linked to antiphospholipid antibodies syndrome (aPL), suggesting PZ-ZPI complex incompetency by Steffano et al. and confirmed by a later study by McColl et al. Kemkes-Matthes et al. found that patients with thromboembolic episodes and factor V Leiden mutations suffered greatly from early thrombosis due to PZ deficiency. Lower levels of plasma PZ have also been linked to chronic inflammatory diseases like Bençet’s disease and ischemic colitis, and acute coronary syndromes (ACS).		In 2002 Gris et al. examined PZ levels in pathologic pregnancies and found that low PZ levels caused extreme insufficiency of the placenta following the mother/fetus circulatory connection. Low PZ levels were also linked to antiphospholipid antibodies syndrome (aPL), suggesting PZ-ZPI complex incompetency by Steffano et al. and confirmed by a later study by McColl et al. Kemkes-Matthes et al. found that patients with thromboembolic episodes and factor V Leiden mutations suffered greatly from early thrombosis due to PZ deficiency. Lower levels of plasma PZ have also been linked to chronic inflammatory diseases like Bençet’s disease and ischemic colitis, and acute coronary syndromes (ACS). [1,6]