Sandbox 7465: Difference between revisions
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==References==<references/> | ==References==<references/> | ||
<ref | <ref>Carle, T. L., Ohnishi, Y. N., Ohnishi, Y. H., Alibhai, I. N., Wilkinson, M. B., Kumar, A. and Nestler, E. J. (2007) Proteasome-dependent and -independent mechanisms for FosB destabilization: identification of FosB degron domains and implications for ΔFosB stability. European Journal of Neuroscience, 25, 3009–3019. doi: 10.1111/j.1460-9568.2007.05575.x</ref> | ||
<ref | <ref>James, K., Ruffle, B. S. (2014) Molecular neurobiology of addiction: what’s all the (∆)FosB about?, The American Journal of Drug and Alcohol Abuse, 40(6), 428-437.</ref> | ||
<ref | <ref>Jorissen, H. J., Ulery, P. G., Henry, L., Gourneni, S., Nestler, E. J., & Rudenko, G. (2007) Dimerization and DNA-binding properties of the transcription factor ΔFosB. Biochemistry, 46(28), 8360-8372.x</ref> | ||
<ref | <ref>Kelz, M. B., Chen, J., Carlezon, W. A., Whisler, K., Gilden, L., Beckmann, A. M., ... & Nestler, E. J. (1999). Expression of the transcription factor ΔFosB in the brain controls sensitivity to cocaine. Nature, 401(6750), 272-276.x</ref> | ||
<ref | <ref>Nestler, E. J. (2008). Transcriptional mechanisms of addiction: role of ΔFosB.Philosophical Transactions of the Royal Society of London B: Biological Sciences, 363(1507), 3245-3255.x</ref> | ||
<ref | <ref>Pitchers, K. K., Vialou, V., Nestler, E. J., Laviolette, S. R., Lehman, M. N., & Coolen, L. M. (2013). Natural and drug rewards act on common neural plasticity mechanisms with ΔFosB as a key mediator. The Journal of Neuroscience,33(8), 3434-3442.x</ref> | ||
<references/> | <references/> | ||
