1vs0

From Proteopedia

(Difference between revisions)
Jump to: navigation, search
(New page: 200px<br /><applet load="1vs0" size="450" color="white" frame="true" align="right" spinBox="true" caption="1vs0, resolution 2.400&Aring;" /> '''Crystal Structure o...)
Line 1: Line 1:
-
[[Image:1vs0.gif|left|200px]]<br /><applet load="1vs0" size="450" color="white" frame="true" align="right" spinBox="true"
+
[[Image:1vs0.gif|left|200px]]<br /><applet load="1vs0" size="350" color="white" frame="true" align="right" spinBox="true"
caption="1vs0, resolution 2.400&Aring;" />
caption="1vs0, resolution 2.400&Aring;" />
'''Crystal Structure of the Ligase Domain from M. tuberculosis LigD at 2.4A'''<br />
'''Crystal Structure of the Ligase Domain from M. tuberculosis LigD at 2.4A'''<br />
==Overview==
==Overview==
-
DNA ligase D (LigD) is a large polyfunctional enzyme involved in, nonhomologous end-joining (NHEJ) in mycobacteria. LigD consists of a, C-terminal ATP-dependent ligase domain fused to upstream polymerase and, phosphoesterase modules. Here we report the 2.4 angstroms crystal, structure of the ligase domain of Mycobacterium LigD, captured as the, covalent ligase-AMP intermediate with a divalent metal in the active site., A chloride anion on the protein surface coordinated by the ribose 3'-OH, and caged by arginine and lysine side chains is a putative mimetic of the, 5'-phosphate at a DNA nick. Structure-guided mutational analysis revealed, distinct requirements for the adenylylation and end-sealing reactions, catalyzed by LigD. We found that a mutation of Mycobacterium LigD that, ablates only ligase activity results in decreased fidelity of NHEJ in vivo, and a strong bias of mutagenic events toward deletions instead of, insertions at the sealed DNA ends. This phenotype contrasts with the, increased fidelity of double-strand break repair in deltaligD cells or in, a strain in which only the polymerase function of LigD is defective. We, surmise that the signature error-prone quality of bacterial NHEJ in vivo, arises from a dynamic balance between the end-remodeling and end-sealing, steps.
+
DNA ligase D (LigD) is a large polyfunctional enzyme involved in nonhomologous end-joining (NHEJ) in mycobacteria. LigD consists of a C-terminal ATP-dependent ligase domain fused to upstream polymerase and phosphoesterase modules. Here we report the 2.4 angstroms crystal structure of the ligase domain of Mycobacterium LigD, captured as the covalent ligase-AMP intermediate with a divalent metal in the active site. A chloride anion on the protein surface coordinated by the ribose 3'-OH and caged by arginine and lysine side chains is a putative mimetic of the 5'-phosphate at a DNA nick. Structure-guided mutational analysis revealed distinct requirements for the adenylylation and end-sealing reactions catalyzed by LigD. We found that a mutation of Mycobacterium LigD that ablates only ligase activity results in decreased fidelity of NHEJ in vivo and a strong bias of mutagenic events toward deletions instead of insertions at the sealed DNA ends. This phenotype contrasts with the increased fidelity of double-strand break repair in deltaligD cells or in a strain in which only the polymerase function of LigD is defective. We surmise that the signature error-prone quality of bacterial NHEJ in vivo arises from a dynamic balance between the end-remodeling and end-sealing steps.
==About this Structure==
==About this Structure==
-
1VS0 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Mycobacterium_tuberculosis Mycobacterium tuberculosis] with ZN, CL and MG as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1VS0 OCA].
+
1VS0 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Mycobacterium_tuberculosis Mycobacterium tuberculosis] with <scene name='pdbligand=ZN:'>ZN</scene>, <scene name='pdbligand=CL:'>CL</scene> and <scene name='pdbligand=MG:'>MG</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1VS0 OCA].
==Reference==
==Reference==
Line 15: Line 15:
[[Category: Akey, D.]]
[[Category: Akey, D.]]
[[Category: Aniukwu, J.]]
[[Category: Aniukwu, J.]]
-
[[Category: Berger, J.M.]]
+
[[Category: Berger, J M.]]
-
[[Category: Glickman, M.S.]]
+
[[Category: Glickman, M S.]]
[[Category: Martins, A.]]
[[Category: Martins, A.]]
[[Category: Shuman, S.]]
[[Category: Shuman, S.]]
-
[[Category: TBSGC, TB.Structural.Genomics.Consortium.]]
+
[[Category: TBSGC, TB Structural Genomics Consortium.]]
[[Category: CL]]
[[Category: CL]]
[[Category: MG]]
[[Category: MG]]
Line 30: Line 30:
[[Category: tbsgc]]
[[Category: tbsgc]]
-
''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Wed Nov 21 05:05:54 2007''
+
''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 15:38:01 2008''

Revision as of 13:38, 21 February 2008


1vs0, resolution 2.400Å

Drag the structure with the mouse to rotate

Crystal Structure of the Ligase Domain from M. tuberculosis LigD at 2.4A

Overview

DNA ligase D (LigD) is a large polyfunctional enzyme involved in nonhomologous end-joining (NHEJ) in mycobacteria. LigD consists of a C-terminal ATP-dependent ligase domain fused to upstream polymerase and phosphoesterase modules. Here we report the 2.4 angstroms crystal structure of the ligase domain of Mycobacterium LigD, captured as the covalent ligase-AMP intermediate with a divalent metal in the active site. A chloride anion on the protein surface coordinated by the ribose 3'-OH and caged by arginine and lysine side chains is a putative mimetic of the 5'-phosphate at a DNA nick. Structure-guided mutational analysis revealed distinct requirements for the adenylylation and end-sealing reactions catalyzed by LigD. We found that a mutation of Mycobacterium LigD that ablates only ligase activity results in decreased fidelity of NHEJ in vivo and a strong bias of mutagenic events toward deletions instead of insertions at the sealed DNA ends. This phenotype contrasts with the increased fidelity of double-strand break repair in deltaligD cells or in a strain in which only the polymerase function of LigD is defective. We surmise that the signature error-prone quality of bacterial NHEJ in vivo arises from a dynamic balance between the end-remodeling and end-sealing steps.

About this Structure

1VS0 is a Single protein structure of sequence from Mycobacterium tuberculosis with , and as ligands. Full crystallographic information is available from OCA.

Reference

Crystal structure and nonhomologous end-joining function of the ligase component of Mycobacterium DNA ligase D., Akey D, Martins A, Aniukwu J, Glickman MS, Shuman S, Berger JM, J Biol Chem. 2006 May 12;281(19):13412-23. Epub 2006 Feb 13. PMID:16476729

Page seeded by OCA on Thu Feb 21 15:38:01 2008

Proteopedia Page Contributors and Editors (what is this?)

OCA

Personal tools